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Denisova Elena1, Makarova Elena2, Savinkova M3
1Laboratory of Physiological Genetics Institute of Cytology and Genetics SB RAS Novosibirsk, RussiaLaboratory of Physiological Genetics Institute of Cytology and Genetics SB RAS Novosibirsk, Russia, elena_nsib@list.ru
2Laboratory of Physiological Genetics Institute of Cytology and Genetics SB RAS Novosibirsk, Russia, enmakarova@gmail.com
3Department of Physiology Novosibirsk State University Novosibirsk, Russia, m.savinkova@g.nsu.ru
Waiting is currently the leader among noncommunicable diseases. Calorie overload causes the development of obesity. It was shown that the prenatal and early postnatal state affects the susceptibility to obesity and can affect taste preferences [1], however, the mechanisms mediating the maternal effect on taste preferences in the offspring are unknown. The adipose tissue leptin hormone may be one of the factors mediating the maternal effect on the phenotype of the offspring. It has been shown that increasing the level of mites in the blood of pregnant women [2, 3] and programming the effects of leptins can be different in the offspring of different sexes. It is not known whether this is due to the positive programmatic effect of maternal leptin with a predisposition to its influence on taste preferences. These studies were supposed to affect pregnancy, muscle metabolism, the frequency of burns caused by diet and taste preferences in offspring of different sexes.
Did the food preferences of the offspring differ with and without leptin
Regardless of leptin administration males and females differed in taste preferences. Females consumed more cookies and less standard chow than males but leptin administration reduced the consumption of cookies in both females and males
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